INT328923

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.18

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Car2, Prkca) cytosol (Car2, Prkca) mitochondrion (Prkca)
lyase activity (Car2) endoplasmic reticulum (Prkca) enzyme binding (Prkca)
Anatomy Link Frequency
B cells 1
Car2 (Mus musculus)
Prkca (Mus musculus)
Pain Link Frequency Relevance Heat
Kinase C 17 100.00 Very High Very High Very High
cINOD 3 5.00 Very Low Very Low Very Low
Neuronal excitability 1 5.00 Very Low Very Low Very Low
depression 1 5.00 Very Low Very Low Very Low
Glutamate 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Shock 2 5.00 Very Low Very Low Very Low
Anxiety Disorder 2 5.00 Very Low Very Low Very Low
Depression 1 5.00 Very Low Very Low Very Low
INFLAMMATION 1 5.00 Very Low Very Low Very Low
Ganglion Cysts 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
While we present evidence in this paper that AA indirectly mediates the reduction in spike frequency produced by EU training, AA has also been reported to act synergistically with diacylglycerol (DAG) and Ca2+ to activate PKC in B cells (Lester et al., 1991), suggesting its involvement in excitatory conditioning (Talk et al., 1997; Muzzio et al., 2001).This potential dual role for AA signaling can be accommodated by several models.
Ca2 Positive_regulation (activate) of PKC in B cells associated with kinase c
1) Confidence 0.00 Published 2010 Journal Frontiers in Behavioral Neuroscience Section Body Doc Link PMC2928666 Disease Relevance 0 Pain Relevance 0.18

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox