INT328946

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Context Info
Confidence 0.43
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 7
Disease Relevance 7.18
Pain Relevance 2.66

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (KIR2DS5)
Anatomy Link Frequency
kidney 1
KIR2DS5 (Homo sapiens)
Pain Link Frequency Relevance Heat
psoriasis 14 98.92 Very High Very High Very High
rheumatoid arthritis 49 98.04 Very High Very High Very High
spinal inflammation 161 97.92 Very High Very High Very High
endometriosis 84 88.00 High High
tolerance 21 35.48 Quite Low
pain pelvic 7 5.00 Very Low Very Low Very Low
Dismenorea 7 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Psoriasis 14 98.92 Very High Very High Very High
Rheumatoid Arthritis 49 98.04 Very High Very High Very High
Graft Vs Host Disease 42 98.04 Very High Very High Very High
Low Back Pain 161 97.92 Very High Very High Very High
Disease 105 97.36 Very High Very High Very High
Leukemia 77 91.44 High High
Reprotox - General 3 35 90.92 High High
Lung Cancer 49 89.16 High High
Cancer 14 88.60 High High
Endometriosis (extended) 84 88.00 High High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
A protective effect of KIR2DS5 gene on susceptibility to psoriasis vulgaris was already described elsewhere [4].
Negative_regulation (effect) of KIR2DS5 associated with psoriasis
1) Confidence 0.43 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2928722 Disease Relevance 0.54 Pain Relevance 0.23
0.19–0.65), whereas a lack of KIR2DS5 and the presence of the HLA-C C2 allotype was associated with ankylosing spondylitis (p?
Negative_regulation (lack) of KIR2DS5 associated with spinal inflammation
2) Confidence 0.43 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2928722 Disease Relevance 1.07 Pain Relevance 0.43
0.25–0.89), whereas lack of KIR2DS5 and presence of C2 seemed to favor rejection (p?
Negative_regulation (lack) of KIR2DS5
3) Confidence 0.43 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2928722 Disease Relevance 0.95 Pain Relevance 0.39
In both instances a decrease in KIR2DS5 frequency was observed.
Negative_regulation (decrease) of KIR2DS5
4) Confidence 0.38 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2928722 Disease Relevance 1.49 Pain Relevance 0.45
However, these results are easily explainable by the protective effect of KIR2DS5 alone.
Negative_regulation (effect) of KIR2DS5
5) Confidence 0.38 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2928722 Disease Relevance 1.27 Pain Relevance 0.38
Therefore, the apparent protective effect of KIR2DS5 described here for kidney rejection may result from this LD.
Negative_regulation (effect) of KIR2DS5 in kidney
6) Confidence 0.37 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2928722 Disease Relevance 0.80 Pain Relevance 0.36
1.28–2.89), whereas a lack of KIR2DS5 and presence of C1 allotype was associated with rheumatoid arthritis (p?
Negative_regulation (lack) of KIR2DS5 associated with rheumatoid arthritis
7) Confidence 0.37 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2928722 Disease Relevance 1.06 Pain Relevance 0.43

General Comments

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