INT329221

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Context Info
Confidence 0.06
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.48
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

oxidoreductase activity (Hmgcr) nucleus (Prkag1) embryo development (Hmgcr)
protein complex (Prkag1) response to stress (Prkag1)
Prkag1 (Mus musculus)
Hmgcr (Mus musculus)
Pain Link Frequency Relevance Heat
cytokine 2 13.68 Low Low
agonist 46 5.00 Very Low Very Low Very Low
Inflammation 6 5.00 Very Low Very Low Very Low
fibrosis 2 5.00 Very Low Very Low Very Low
cINOD 2 5.00 Very Low Very Low Very Low
ischemia 2 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
tolerance 2 5.00 Very Low Very Low Very Low
Kinase C 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hyperlipidemia 4 94.44 High High
Stress 14 65.92 Quite High
Pathologic Processes 4 48.16 Quite Low
Cardiovascular Disease 4 46.24 Quite Low
Diabetes Mellitus 16 44.56 Quite Low
Insulin Resistance 30 44.04 Quite Low
Increased Venous Pressure Under Development 10 43.76 Quite Low
Coronary Artery Disease 2 42.16 Quite Low
Apoptosis 2 39.44 Quite Low
Injury 2 34.48 Quite Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Moreover, AMPK inhibits hepatic cholesterol synthesis by inhibiting HMG-CoA reductase, a rate-controlling enzyme of the mevalonate pathway.
AMPK Negative_regulation (inhibiting) of HMG-CoA reductase
1) Confidence 0.06 Published 2010 Journal PPAR Research Section Body Doc Link PMC2929615 Disease Relevance 0.21 Pain Relevance 0
AMPK inhibits HMG-CoA reductase activity by phosphorylation, which reduces cholesterol levels (Figure 2) [47].
AMPK Negative_regulation (inhibits) of HMG-CoA reductase
2) Confidence 0.06 Published 2010 Journal PPAR Research Section Body Doc Link PMC2929615 Disease Relevance 0.27 Pain Relevance 0

General Comments

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