INT329296

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Context Info
Confidence 0.39
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 4
Disease Relevance 1.72
Pain Relevance 1.77

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleolus (Aff3) nucleus (Aff3)
Aff3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Calcitonin gene-related peptide 8 98.90 Very High Very High Very High
qutenza 94 98.72 Very High Very High Very High
rheumatoid arthritis 56 97.00 Very High Very High Very High
agonist 4 83.72 Quite High
Adalimumab 4 80.28 Quite High
Infliximab 6 79.72 Quite High
Etanercept 6 79.24 Quite High
Pain 2 64.76 Quite High
antagonist 12 50.00 Quite Low
anesthesia 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Rheumatoid Arthritis 58 97.00 Very High Very High Very High
Disease 14 84.60 Quite High
Increased Venous Pressure Under Development 52 84.08 Quite High
Injury 44 78.56 Quite High
Necrosis 6 75.00 Quite High
Cancer 6 75.00 Quite High
Acid Reflux 10 63.92 Quite High
Diabetes Mellitus 8 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 2 5.00 Very Low Very Low Very Low
Disease Susceptibility 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In vitro studies have failed to document a direct interaction between Laf and TRPV1 [12, 14], whereas Laf potentiates Cap-induced CGRP release from the gastric mucosa [14], suggesting that Laf sensitizes TRPV1 or interacts at alternate components of the capsaicin pathway.
Laf Binding (interaction) of associated with qutenza and calcitonin gene-related peptide
1) Confidence 0.39 Published 2010 Journal Dig Dis Sci Section Body Doc Link PMC2958262 Disease Relevance 0.34 Pain Relevance 0.69
In vitro studies have failed to document a direct interaction between Laf and TRPV1 [12, 14], whereas Laf potentiates Cap-induced CGRP release from the gastric mucosa [14], suggesting that Laf sensitizes TRPV1 or interacts at alternate components of the capsaicin pathway.
Laf Binding (interacts) of associated with qutenza and calcitonin gene-related peptide
2) Confidence 0.30 Published 2010 Journal Dig Dis Sci Section Body Doc Link PMC2958262 Disease Relevance 0.40 Pain Relevance 0.67
Two variants mapping to AFF3 and one to the CD226 locus demonstrated statistically significant evidence for association under an additive model (table 3) (AFF3: rs10865035, allele G coefficient ?
AFF3 Binding (mapping) of
3) Confidence 0.26 Published 2010 Journal Annals of the Rheumatic Diseases Section Body Doc Link PMC2930850 Disease Relevance 0.15 Pain Relevance 0.07
In the combined analysis, SNPs mapping to AFF3 and CD226 had a statistically significant association with the response to anti-TNF treatment under an additive model.
AFF3 Binding (association) of
4) Confidence 0.25 Published 2010 Journal Annals of the Rheumatic Diseases Section Abstract Doc Link PMC2930850 Disease Relevance 0.84 Pain Relevance 0.34

General Comments

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