INT329317

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Context Info
Confidence 0.02
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.05
Pain Relevance 0.12

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (ACE, KNG1) extracellular region (ACE, KNG1) plasma membrane (ACE, KNG1)
endosome (ACE) peptidase activity (ACE)
ACE (Homo sapiens)
KNG1 (Homo sapiens)
Pain Link Frequency Relevance Heat
bradykinin 1 100.00 Very High Very High Very High
ischemia 6 85.60 High High
fibrosis 20 62.68 Quite High
Inflammation 14 48.32 Quite Low
cytokine 10 36.60 Quite Low
cva 3 5.00 Very Low Very Low Very Low
Angina 2 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
Calcium channel 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Heart Rate Under Development 12 98.28 Very High Very High Very High
Left Ventricular Dysfunction 1 97.32 Very High Very High Very High
Cv Unclassified Under Development 6 88.44 High High
Chagas Cardiomyopathy 34 84.00 Quite High
Necrosis 12 77.96 Quite High
Congenital Anomalies 8 67.80 Quite High
Fibrosis 21 62.68 Quite High
Sprains And Strains 11 57.52 Quite High
Acute Myocarditis 2 54.16 Quite High
Infection 26 51.60 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The mechanism of action of captopril, an ACEI, involves suppression of angiotensin II, a potent vasoconstrictor, and increased levels of bradykinin through the inhibition of kininase II, which induces NO release in ECs and stimulates the production of prostacyclin, a vasodilating prostaglandin [117].
ACEI Regulation (involves) of Positive_regulation (increased) of bradykinin associated with bradykinin
1) Confidence 0.02 Published 2010 Journal PLoS Neglected Tropical Diseases Section Body Doc Link PMC2930857 Disease Relevance 1.05 Pain Relevance 0.12

General Comments

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