INT330133

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 3
Speculated 0
Reported most in Body
Documents 1
Total Number 3
Disease Relevance 0.63
Pain Relevance 0.98

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

RT1-O2-ps (Rattus norvegicus)
Pain Link Frequency Relevance Heat
antagonist 39 99.78 Very High Very High Very High
B1 receptor 273 99.56 Very High Very High Very High
agonist 45 81.68 Quite High
Spinal cord 33 59.84 Quite High
allodynia 63 50.00 Quite Low
bradykinin 39 38.88 Quite Low
diabetic neuropathy 3 5.72 Low Low
Pain 18 5.00 Very Low Very Low Very Low
withdrawal 15 5.00 Very Low Very Low Very Low
Inflammation 12 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 99 95.04 Very High Very High Very High
Diabetes Mellitus 51 89.32 High High
Pressure And Volume Under Development 57 82.60 Quite High
Neuropathic Pain 78 50.00 Quite Low
Insulin Resistance 39 50.00 Quite Low
Hyperglycemia 3 9.48 Low Low
Diabetic Neuropathy 3 5.72 Low Low
Pain 15 5.00 Very Low Very Low Very Low
Hypertension 12 5.00 Very Low Very Low Very Low
INFLAMMATION 12 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The latter treatment with the B1R antagonist failed to alter O2•?
Neg (failed) Regulation (alter) of O2 associated with antagonist and b1 receptor
1) Confidence 0.01 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2935380 Disease Relevance 0.07 Pain Relevance 0.33
This contrasts with the acute treatment with SSR240612 which did not affect O2•?
Neg (not) Regulation (affect) of O2
2) Confidence 0.01 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2935380 Disease Relevance 0.48 Pain Relevance 0.25
SSR240612 failed, however, to alter basal O2•?
Neg (failed) Regulation (alter) of O2
3) Confidence 0.01 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2935380 Disease Relevance 0.08 Pain Relevance 0.40

General Comments

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