INT330226

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 1
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.07
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleus (Prkaa2, Trib3) nucleoplasm (Prkaa2) enzyme binding (Trib3)
protein binding, bridging (Prkaa2) protein complex (Prkaa2) response to stress (Prkaa2)
Anatomy Link Frequency
STO 2
Prkaa2 (Rattus norvegicus)
Trib3 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
imagery 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
Kinase C 1 5.00 Very Low Very Low Very Low
qutenza 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 22 71.92 Quite High
Hepatocellular Cancer 1 45.36 Quite Low
Parkinson's Disease 2 5.00 Very Low Very Low Very Low
Immunization 1 5.00 Very Low Very Low Very Low
Diabetes Mellitus 1 5.00 Very Low Very Low Very Low
Hypoxia 1 5.00 Very Low Very Low Very Low
Targeted Disruption 1 5.00 Very Low Very Low Very Low
Hypertrophy 1 5.00 Very Low Very Low Very Low
Cancer 1 5.00 Very Low Very Low Very Low
Shock 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
To examine whether activation of AMPK by agents that acted in an AMP-independent manner required calmodulin-dependent protein kinase kinases (CaMKKs), we utilized the CaMKK inhibitor, STO-609 (FigureĀ 7A).
Spec (20132) Positive_regulation (activation) of AMPK in STO Positive_regulation (required) of kinase
1) Confidence 0.00 Published 2010 Journal Cell Metabolism Section Body Doc Link PMC2935965 Disease Relevance 0.07 Pain Relevance 0

General Comments

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