INT330668

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Context Info
Confidence 0.05
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.10
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

structural molecule activity (LMNB2) nucleus (LMNB2) molecular_function (LMNB2)
biological_process (LMNB2)
Anatomy Link Frequency
lamina 1
LMNB2 (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Herpes Virus Infection 1 99.80 Very High Very High Very High
Retinoblastoma 51 5.00 Very Low Very Low Very Low
Infection 11 5.00 Very Low Very Low Very Low
Viral Infection 10 5.00 Very Low Very Low Very Low
Cancer 6 5.00 Very Low Very Low Very Low
Epstein-barr Virus 4 5.00 Very Low Very Low Very Low
Stress 2 5.00 Very Low Very Low Very Low
Herpes Zoster 2 5.00 Very Low Very Low Very Low
Leukemia 2 5.00 Very Low Very Low Very Low
Apoptosis 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Although experiments of this type are often cited as evidence that CHPK-mediated lamin A/C phosphorylation directly leads to lamina disruption, we note that they cannot rule out other mechanisms for lamina disruption upon CHPK expression or herpesvirus infection (such as lamin B or lamin receptor phosphorylation) in addition to or instead of lamin A/C phosphorylation.


Phosphorylation (phosphorylation) of lamin B in lamina associated with herpes virus infection
1) Confidence 0.05 Published 2010 Journal PLoS Pathogens Section Body Doc Link PMC2936540 Disease Relevance 0.10 Pain Relevance 0

General Comments

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