INT331943

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Context Info
Confidence 0.04
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.70
Pain Relevance 0.25

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transferase activity, transferring glycosyl groups (Tymp)
Tymp (Mus musculus)
Pain Link Frequency Relevance Heat
agonist 40 99.84 Very High Very High Very High
Kinase C 2 95.72 Very High Very High Very High
Angina 2 73.96 Quite High
antagonist 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 32 77.28 Quite High
Cv General 3 Under Development 2 73.96 Quite High
Coronary Artery Disease 2 72.88 Quite High
Increased Venous Pressure Under Development 4 63.96 Quite High
Hypertension 2 63.16 Quite High
Hypertrophy 2 59.72 Quite High
Adhesions 2 57.40 Quite High
Apoptosis 2 55.28 Quite High
Cardiovascular Disease 2 51.36 Quite High
Hypersensitivity 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
H2O2 treatment results in potentiation of the increase to intracellular calcium concentrations in response to TP?
Regulation (response) of TP
1) Confidence 0.04 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2939892 Disease Relevance 0.07 Pain Relevance 0.09
Regulation of TP signaling itself is similarly complex, with multiple kinase-mediated pathways implicated in receptor desensitization, while oligomerization of the distinct TP splice variant products has also been implicated in modulating function [8], [9].
Regulation (Regulation) of TP
2) Confidence 0.03 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2939892 Disease Relevance 0.63 Pain Relevance 0.16

General Comments

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