INT332018

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.23
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.87
Pain Relevance 0.21

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Gopc) Golgi apparatus (Gopc) plasma membrane (Gopc)
cytoplasm (Gopc)
Anatomy Link Frequency
apoptotic cells 1
C1qa (Mus musculus)
Gopc (Mus musculus)
Pain Link Frequency Relevance Heat
ischemia 20 92.84 High High
Inflammation 12 78.24 Quite High
antagonist 5 5.00 Very Low Very Low Very Low
Inflammatory response 2 5.00 Very Low Very Low Very Low
isoflurane 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 1 98.44 Very High Very High Very High
Cv Unclassified Under Development 16 92.84 High High
Injury 37 89.52 High High
Myocarditis 2 78.24 Quite High
Coronary Artery Disease 7 73.80 Quite High
Hypoxia 1 50.52 Quite High
Death 4 47.64 Quite Low
INFLAMMATION 12 5.00 Very Low Very Low Very Low
Neutropenia 9 5.00 Very Low Very Low Very Low
Myocardial Infarction 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The complement subcomponent C1q can initiate classical pathway activation by recognition of these antigen-antibody complexes (Figure 1 (Fig. 1)); however, C1q can also recognize apoptotic cells in the absence of antibodies [7], [8], [9].
C1q Binding (recognition) of Fig in apoptotic cells associated with apoptosis
1) Confidence 0.23 Published 2010 Journal GMS German Medical Science Section Body Doc Link PMC2940219 Disease Relevance 0.87 Pain Relevance 0.21

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox