INT333531

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Context Info
Confidence 0.60
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.69
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

small molecule metabolic process (CACNA1C) plasma membrane (CACNA1C) transmembrane transport (CACNA1C)
cytoplasm (CACNA1C)
CACNA1C (Homo sapiens)
Pain Link Frequency Relevance Heat
action potential duration 20 49.68 Quite Low
antagonist 2 45.60 Quite Low
Action potential 40 5.00 Very Low Very Low Very Low
lidocaine 9 5.00 Very Low Very Low Very Low
addiction 3 5.00 Very Low Very Low Very Low
agonist 3 5.00 Very Low Very Low Very Low
Calcium channel 2 5.00 Very Low Very Low Very Low
tetrodotoxin 1 5.00 Very Low Very Low Very Low
sodium channel 1 5.00 Very Low Very Low Very Low
Antihistamine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Syndrome 8 95.36 Very High Very High Very High
Autism 1 86.16 High High
Symphalangism 1 84.72 Quite High
Heart Rate Under Development 4 76.00 Quite High
Cv General 2 Under Development 1 67.56 Quite High
Arrhythmogenic Right Ventricular Dysplasia 3 63.76 Quite High
Cardiovascular Disorder Under Development 4 50.00 Quite Low
Arrhythmia Under Development 9 5.00 Very Low Very Low Very Low
Toxicity 4 5.00 Very Low Very Low Very Low
Arrhythmias 2 Under Development 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
It was found that the origin of this multi-system disorder was a de novo missense mutation affecting the Cav1.2 gene (one of the aliases commonly used for the CACNA1C gene).
Regulation (affecting) of Cav1.2
1) Confidence 0.60 Published 2010 Journal Philosophical transactions. Series A, Mathematical, physical, and engineering sciences Section Body Doc Link PMC2944395 Disease Relevance 0.69 Pain Relevance 0

General Comments

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