INT334158

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Context Info
Confidence 0.10
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.85
Pain Relevance 0.12

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (DTNA) plasma membrane (DTNA) cytoplasm (DTNA)
DTNA (Homo sapiens)
Pain Link Frequency Relevance Heat
fibrosis 7 94.24 High High
imagery 2 48.80 Quite Low
positron emission tomography 1 47.56 Quite Low
cva 5 24.08 Low Low
Angina 2 5.00 Very Low Very Low Very Low
drug abuse 1 5.00 Very Low Very Low Very Low
Morphine 1 5.00 Very Low Very Low Very Low
alcohol 1 5.00 Very Low Very Low Very Low
beta blocker 1 5.00 Very Low Very Low Very Low
Osteoarthritis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Heart Defects 33 100.00 Very High Very High Very High
Muscle Disease 2 96.96 Very High Very High Very High
Fibrosis 6 94.24 High High
Congenital Structural Myopathies 1 91.48 High High
Frailty 1 90.48 High High
Barth Syndrome 2 89.08 High High
Myocardial Infarction 5 85.04 High High
Mitochondrial Myopathies 1 83.44 Quite High
Disease 1 72.64 Quite High
Cardiomyopathy 7 71.80 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Genetic mutations, especially the alpha-dystrobrevin gene, Cypher/ZASP and gene G4.5 of the Xq28 chromosome region and loss of the cardiac-specific gene CSX have been well described in the literature as causes of LVNC [15-17].
Positive_regulation (causes) of LVNC associated with heart defects
1) Confidence 0.10 Published 2010 Journal Int Arch Med Section Body Doc Link PMC2945326 Disease Relevance 1.85 Pain Relevance 0.12

General Comments

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