INT335592

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.19
Pain Relevance 0.74

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Akt1) aging (Akt1) enzyme binding (Akt1)
carbohydrate metabolic process (Akt1) cytoplasm (Akt1) cytosol (Akt1)
Ltp (Mus musculus)
Akt1 (Mus musculus)
Pain Link Frequency Relevance Heat
long-term potentiation 83 100.00 Very High Very High Very High
Anterior cingulate cortex 78 82.56 Quite High
nMDA receptor 11 5.00 Very Low Very Low Very Low
Pyramidal cell 5 5.00 Very Low Very Low Very Low
Hippocampus 4 5.00 Very Low Very Low Very Low
amygdala 2 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
Neurotransmitter 1 5.00 Very Low Very Low Very Low
halothane 1 5.00 Very Low Very Low Very Low
Glutamate 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 46 72.48 Quite High
Anxiety Disorder 3 43.00 Quite Low
Pain 2 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Thus, LTP is divided into two phases such as early-phase and late-phase LTP; early-phase LTP depends on the activation of kinases and phosphatases, while late-phase LTP depends on new protein synthesis.
LTP Positive_regulation (depends) of Positive_regulation (activation) of kinases associated with long-term potentiation
1) Confidence 0.01 Published 2010 Journal Mol Brain Section Body Doc Link PMC2949850 Disease Relevance 0.19 Pain Relevance 0.74

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox