INT336132

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.38
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (ANG) DNA binding (ANG) signal transducer activity (AGTR1)
extracellular space (ANG) extracellular region (ANG) cell death (ANG)
Anatomy Link Frequency
heart 1
AGTR1 (Homo sapiens)
ANG (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 1 98.32 Very High Very High Very High
fibrosis 3 5.00 Very Low Very Low Very Low
withdrawal 3 5.00 Very Low Very Low Very Low
Inflammation 3 5.00 Very Low Very Low Very Low
ischemia 2 5.00 Very Low Very Low Very Low
Bioavailability 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
agonist 1 5.00 Very Low Very Low Very Low
imagery 1 5.00 Very Low Very Low Very Low
headache 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Increased Venous Pressure Under Development 8 96.74 Very High Very High Very High
Renal Disease 8 96.32 Very High Very High Very High
Myocardial Infarction 22 96.02 Very High Very High Very High
Hypertension 34 95.72 Very High Very High Very High
Hypertrophy 1 33.24 Quite Low
Diabetes Mellitus 23 5.00 Very Low Very Low Very Low
Obesity 10 5.00 Very Low Very Low Very Low
Pressure Volume 2 Under Development 7 5.00 Very Low Very Low Very Low
Hyperkalemia 6 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Pharmacologic blockade of the RAAS has proven to be an effective therapeutic strategy in the treatment of several cardiovascular disorders, including hypertension, heart failure, renal disease, and atherosclerosis.4 Until now, in clinical practice, the drug classes that provided RAAS inhibition included ACE inhibitors (ACEIs), which block the conversion of Ang I to Ang II, angiotensin receptor blockers (ARBs), which interfere with Ang II binding to its Type 1 receptors, and aldosterone antagonists, which inhibit aldosterone action via the mineralocorticoid receptor (MR) receptor.
angiotensin receptor Binding (binding) of Ang in heart associated with antagonist, renal disease, hypertension, increased venous pressure under development and myocardial infarction
1) Confidence 0.00 Published 2010 Journal Vascular Health and Risk Management Section Body Doc Link PMC2952455 Disease Relevance 0.38 Pain Relevance 0.05

General Comments

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