INT336579

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Context Info
Confidence 0.15
First Reported 2010
Last Reported 2010
Negated 0
Speculated 1
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 1.94
Pain Relevance 0.05

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (HCRTR1) signal transducer activity (HCRTR1)
HCRTR1 (Homo sapiens)
Pain Link Frequency Relevance Heat
Neuropeptide 4 91.56 High High
depression 2 5.00 Very Low Very Low Very Low
opiate 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hypersomnia 76 99.56 Very High Very High Very High
Sleep Disorders 174 99.12 Very High Very High Very High
Autoimmune Disease 4 85.48 High High
Idiopathic Hypersomnolence 50 73.52 Quite High
Disease 18 25.00 Low Low
Obesity 14 5.00 Very Low Very Low Very Low
Hallucination 10 5.00 Very Low Very Low Very Low
Ehlers-danlos Syndrome 8 5.00 Very Low Very Low Very Low
Sleep Paralysis 8 5.00 Very Low Very Low Very Low
Disease Progression 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Because any autoAbs exist as a free fraction and as immune complexes, it is possible that relative amount of free and complexed autoAbs against hypocretin peptides may participate in the regulation of hypocretin availability and therefore can be associated with sleep/wake dysregulation.
Spec (may) Regulation (participate) of Regulation (regulation) of hypocretin
1) Confidence 0.15 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2954157 Disease Relevance 0.96 Pain Relevance 0.05
However, the mechanism responsible for alteration of the hypocretin system in narcolepsy-cataplexy and its relevance to other central hypersomnias remain unknown.
Regulation (responsible) of Regulation (alteration) of hypocretin associated with hypersomnia and sleep disorders
2) Confidence 0.13 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2954157 Disease Relevance 0.98 Pain Relevance 0

General Comments

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