INT336933

Context Info Confidence 0.07 First Reported 2010 Last Reported 2010 Negated 0 Speculated 0 Reported most in Body Documents 1 Total Number 2 Disease Relevance 2.30 Pain Relevance 0.31

transferase activity, transferring glycosyl groups (Ugcg) Golgi apparatus (Ugcg) endoplasmic reticulum (Nr3c2) nucleus (Nr3c2) cytoplasm (Nr3c2)

Nr3c2 (Rattus norvegicus) Ugcg (Rattus norvegicus)

Pain Link Frequency Relevance Heat Inflammation 8 96.24 corticosteroid 4 90.64 cytokine 2 63.36 Inflammatory response 2 37.16 isoflurane 6 5.00 Analgesic 2 5.00 anesthesia 2 5.00 Somatosensory cortex 2 5.00

Disease Link Frequency Relevance Heat INFLAMMATION 10 96.24 Stroke 54 94.20 Death 4 89.28 Injury 2 88.52 Stress 102 88.40 Shock 2 80.08 Apoptosis 2 71.60 Disease 2 55.80 Pressure And Volume Under Development 4 49.12 Overdose 2 5.00

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Key:

Protein

Mutation

Event

Anatomy

Negation

Speculation

Pain term

Disease term

The mechanism of action of GCs is through its binding with the type I mineralocorticoid receptor (MR) and the type II GR [45].

MR Binding (binding) of GCs

1) Confidence 0.07 Published 2010 Journal Exp Transl Stroke Med Section Body Doc Link PMC2954925 Disease Relevance 1.15 Pain Relevance 0.16

The mechanism of action of GCs is through its binding with the type I mineralocorticoid receptor (MR) and the type II GR [45].

type I mineralocorticoid receptor Binding (binding) of GCs

2) Confidence 0.07 Published 2010 Journal Exp Transl Stroke Med Section Body Doc Link PMC2954925 Disease Relevance 1.15 Pain Relevance 0.16

General Comments

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