INT338056

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Context Info
Confidence 0.30
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.15

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Ctsg, F2rl3) signal transduction (F2rl3) peptidase activity (Ctsg)
extracellular space (Ctsg) signal transducer activity (F2rl3)
Anatomy Link Frequency
neurons 1
Ctsg (Mus musculus)
F2rl3 (Mus musculus)
Pain Link Frequency Relevance Heat
Nerve growth factor 14 85.52 High High
agonist 14 72.84 Quite High
bradykinin 4 71.00 Quite High
Inflammatory mediators 3 49.28 Quite Low
Calcitonin gene-related peptide 20 5.00 Very Low Very Low Very Low
nociceptor 16 5.00 Very Low Very Low Very Low
qutenza 14 5.00 Very Low Very Low Very Low
Neuropeptide 12 5.00 Very Low Very Low Very Low
Inflammation 12 5.00 Very Low Very Low Very Low
imagery 7 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Nociception 21 50.00 Quite Low
INFLAMMATION 13 48.88 Quite Low
Pain 6 5.00 Very Low Very Low Very Low
Hyperalgesia 3 5.00 Very Low Very Low Very Low
Targeted Disruption 3 5.00 Very Low Very Low Very Low
Injury 3 5.00 Very Low Very Low Very Low
Coagulation Disorder 2 5.00 Very Low Very Low Very Low
Neurogenic Inflammation 2 5.00 Very Low Very Low Very Low
Rupture 2 5.00 Very Low Very Low Very Low
Sprains And Strains 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Cathepsin G, which preferentially activates PAR4 over PAR1, caused translocation in 11.8 ± 2.3% of neurons.
Cathepsin G Positive_regulation (activates) of PAR4 in neurons
1) Confidence 0.30 Published 2010 Journal Mol Pain Section Body Doc Link PMC2956715 Disease Relevance 0 Pain Relevance 0.15

General Comments

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