INT339173

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Context Info
Confidence 0.26
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 2
Total Number 2
Disease Relevance 1.83
Pain Relevance 0.20

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

AREG (Homo sapiens)
Pain Link Frequency Relevance Heat
Inflammation 35 99.12 Very High Very High Very High
rheumatoid arthritis 3 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
Neuropathic pain 1 5.00 Very Low Very Low Very Low
depression 1 5.00 Very Low Very Low Very Low
alcohol 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 36 99.12 Very High Very High Very High
Autoimmune Disease 32 97.68 Very High Very High Very High
Takayasu Arteritis 50 96.68 Very High Very High Very High
Hypertrophy 1 96.00 Very High Very High Very High
Skin Cancer 1 94.08 High High
Breast Cancer 24 89.32 High High
Cancer 80 83.52 Quite High
Tumor Necrosis Factor Receptor-associated Periodic Syndrome 1 70.32 Quite High
Colon Cancer 1 62.72 Quite High
Lobular Carcinoma 1 56.76 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The up-regulation of amphiregulin (AREG), which is a member of the epidermal growth factor family, may be related to the ventricular hypertrophy often observed in TA patients.
Regulation (regulation) of amphiregulin associated with takayasu arteritis and hypertrophy
1) Confidence 0.26 Published 2010 Journal DNA Research: An International Journal for Rapid Publication of Reports on Genes and Genomes Section Body Doc Link PMC2993541 Disease Relevance 1.12 Pain Relevance 0.20
The 4q13.3 gain/amplification targeted 20 genes, including those encoding EGFR ligands EPEG, EREG, AREG and BTC, which could play a critical role in oncogenesis [54].
Regulation (targeted) of AREG
2) Confidence 0.06 Published 2010 Journal BMC Cancer Section Body Doc Link PMC2958950 Disease Relevance 0.71 Pain Relevance 0

General Comments

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