INT340171

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Context Info
Confidence 0.13
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0.29
Pain Relevance 0.17

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Avpr1a) plasma membrane (Avpr1a) response to stress (Avpr1a)
signal transducer activity (Avpr1a)
Anatomy Link Frequency
P19 2
Avpr1a (Mus musculus)
Pain Link Frequency Relevance Heat
antagonist 32 99.76 Very High Very High Very High
Inflammation 2 11.68 Low Low
imagery 4 5.00 Very Low Very Low Very Low
Potency 2 5.00 Very Low Very Low Very Low
Dismenorea 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Embryonal Carcinoma 46 98.36 Very High Very High Very High
INFLAMMATION 2 11.68 Low Low
Natriuresis 6 5.00 Very Low Very Low Very Low
Apoptosis 4 5.00 Very Low Very Low Very Low
Infarction 2 5.00 Very Low Very Low Very Low
Dysmenorrhea 2 5.00 Very Low Very Low Very Low
Hyperplasia 2 5.00 Very Low Very Low Very Low
Repression 2 5.00 Very Low Very Low Very Low
Adhesions 2 5.00 Very Low Very Low Very Low
Osteoporosis 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
OT binding sites disclosed in the V1aR model are in total agreement with those reported by ?
Negative_regulation (disclosed) of Gene_expression (model) of V1aR
1) Confidence 0.13 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2964328 Disease Relevance 0 Pain Relevance 0.03
We observed that both V1aR antagonist as well as V1aR silencing partially blocked the cardiomyogenic effects of OT-GKR in EC P19 cells.
Negative_regulation (blocked) of Gene_expression (silencing) of V1aR in P19 associated with antagonist and embryonal carcinoma
2) Confidence 0.13 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2964328 Disease Relevance 0.29 Pain Relevance 0.14

General Comments

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