INT340201

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Context Info
Confidence 0.49
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.34
Pain Relevance 0.38

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

signal transduction (Htr1a) plasma membrane (Htr1a) signal transducer activity (Htr1a)
Anatomy Link Frequency
neurons 2
Htr1a (Mus musculus)
Pain Link Frequency Relevance Heat
5HT 7 100.00 Very High Very High Very High
medulla 15 92.40 High High
Raphe 4 89.72 High High
fluoxetine 32 56.24 Quite High
Serotonin 6 5.00 Very Low Very Low Very Low
antidepressant 5 5.00 Very Low Very Low Very Low
Raphe magnus 2 5.00 Very Low Very Low Very Low
depression 1 5.00 Very Low Very Low Very Low
Locus ceruleus 1 5.00 Very Low Very Low Very Low
Neurotransmitter 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Acidosis 57 81.36 Quite High
Sudden Infant Death Syndrome 6 5.00 Very Low Very Low Very Low
Targeted Disruption 5 5.00 Very Low Very Low Very Low
Syndrome 4 5.00 Very Low Very Low Very Low
Rare Diseases 3 5.00 Very Low Very Low Very Low
Death 2 5.00 Very Low Very Low Very Low
Hypoventilation 2 5.00 Very Low Very Low Very Low
Hypoxia 1 5.00 Very Low Very Low Very Low
Congenital Anomalies 1 5.00 Very Low Very Low Very Low
Hypercapnia 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Indeed, the 5-HT neurons play a crucial role in respiratory function and dysfunction [14]: they are intrinsically chemosensitive [12], synaptically contact the respiratory neurons [15] and exert a facilitatory modulation on the RRG via a release of endogenous 5HT and activation of 5-HT1A receptors [16], [36].
Positive_regulation (activation) of Localization (release) of 5-HT1A in neurons associated with 5ht
1) Confidence 0.49 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2964329 Disease Relevance 0.34 Pain Relevance 0.38

General Comments

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