INT340238

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Context Info
Confidence 0.02
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.40
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Tnfsf11) Golgi apparatus (Fig4) endoplasmic reticulum (Fig4)
intracellular (Tnfsf11) response to stress (Tnfsf11) pigmentation (Fig4)
Tnfsf11 (Rattus norvegicus)
Fig4 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
antagonist 5 50.00 Quite Low
isoflurane 1 25.80 Quite Low
Potency 5 5.00 Very Low Very Low Very Low
Bioavailability 5 5.00 Very Low Very Low Very Low
agonist 4 5.00 Very Low Very Low Very Low
Inflammation 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
dexamethasone 1 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Respiratory Syncytial Virus 116 91.80 High High
Hypercalcemia 14 69.88 Quite High
Overdose 1 38.20 Quite Low
Body Weight 1 28.40 Quite Low
Cancer 9 5.00 Very Low Very Low Very Low
Multiple Myeloma 6 5.00 Very Low Very Low Very Low
Fibromyalgia 6 5.00 Very Low Very Low Very Low
Osteoporosis 5 5.00 Very Low Very Low Very Low
Toxicity 4 5.00 Very Low Very Low Very Low
Apoptosis 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
When compared with cultures treated with rhM-CSF alone, rhRANKL induced the formation of numerous TRAP+ multinucleated cells (Fig. 1a, b), increased the levels of TRAP activity in media (Fig. 1c), and strongly induced the expression of cathepsin K (Fig. 1d), all known to be hallmarks of OC differentiation.
rhRANKL Positive_regulation (induced) of Gene_expression (expression) of Fig
1) Confidence 0.02 Published 2010 Journal Calcif Tissue Int Section Body Doc Link PMC2964465 Disease Relevance 0.40 Pain Relevance 0

General Comments

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