INT342227

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Context Info
Confidence 0.03
First Reported 2010
Last Reported 2010
Negated 1
Speculated 0
Reported most in Body
Documents 1
Total Number 4
Disease Relevance 1.06
Pain Relevance 0.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

lipid binding (Ppara) nucleus (Ppara) DNA binding (Ppara)
lipid metabolic process (Ppara)
TRNAI1 (Homo sapiens)
Ppara (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 36 94.44 High High
cytokine 4 34.12 Quite Low
agonist 8 5.00 Very Low Very Low Very Low
anesthesia 4 5.00 Very Low Very Low Very Low
alcohol 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 36 94.44 High High
Necrosis 28 93.76 High High
Sprains And Strains 64 85.92 High High
Cancer 4 79.64 Quite High
Toxicity 24 74.20 Quite High
Stress 4 68.00 Quite High
Hepatomegaly 16 33.04 Quite Low
Hypertrophy 8 27.36 Quite Low
Injury 24 10.72 Low Low
Hepatotoxicity 8 9.28 Low Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
mice, but 2,000 ppm TRI significantly increased mouse PPAR?
TRI Positive_regulation (increased) of PPAR
1) Confidence 0.03 Published 2010 Journal Environ Health Perspect Section Body Doc Link PMC2974693 Disease Relevance 0.22 Pain Relevance 0
TRI induced hepatic PPAR?
TRI Positive_regulation (induced) of PPAR
2) Confidence 0.03 Published 2010 Journal Environ Health Perspect Section Body Doc Link PMC2974693 Disease Relevance 0.08 Pain Relevance 0
No differences were observed in TRI-mediated induction of hepatic PPAR?
TRI Positive_regulation (induction) of PPAR
3) Confidence 0.03 Published 2010 Journal Environ Health Perspect Section Abstract Doc Link PMC2974693 Disease Relevance 0.39 Pain Relevance 0.11
mice, but TRI exposure did not induce PPAR?
TRI Neg (not) Positive_regulation (induce) of PPAR
4) Confidence 0.03 Published 2010 Journal Environ Health Perspect Section Body Doc Link PMC2974693 Disease Relevance 0.36 Pain Relevance 0

General Comments

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