INT342459

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.59
Pain Relevance 0.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (CXCR3, CRTC1) cytoplasm (CXCR3, CRTC1) nucleolus (CRTC1)
cell adhesion (CXCR3) nucleus (CRTC1) signal transducer activity (CXCR3)
Anatomy Link Frequency
T-cell 1
CXCR3 (Homo sapiens)
CRTC1 (Homo sapiens)
Pain Link Frequency Relevance Heat
chemokine 8 59.04 Quite High
cytokine 12 53.36 Quite High
Inflammatory response 4 50.72 Quite High
imagery 60 50.00 Quite Low
Inflammation 38 34.12 Quite Low
COX2 1 32.80 Quite Low
cva 11 5.00 Very Low Very Low Very Low
rheumatoid arthritis 6 5.00 Very Low Very Low Very Low
metalloproteinase 5 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 6 97.24 Very High Very High Very High
Hyperplasia 1 91.84 High High
Injury 2 71.96 Quite High
Atherosclerosis 102 68.44 Quite High
INFLAMMATION 40 50.72 Quite High
Stress 5 28.88 Quite Low
Cardiovascular Disease 19 17.92 Low Low
Thrombosis 21 5.00 Very Low Very Low Very Low
Atherosclerotic Plaque 19 5.00 Very Low Very Low Very Low
Disease 15 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
These results strongly indicate that CXCR3-dependent activation of mTORC1 directly links stimulation of the Th1 lymphocytesÂ’ immune system with the proliferative response of intimal cells in vascular remodelling: a process that is followed by expression of activation markers on T-cell surface such as the IL-2 receptor, CD25-CD122-CD132 heterotrimer [50].
CXCR3-dependent Positive_regulation (activation) of mTORC1 in T-cell
1) Confidence 0.00 Published 2010 Journal Eur J Nucl Med Mol Imaging Section Body Doc Link PMC2975909 Disease Relevance 0.59 Pain Relevance 0.11

General Comments

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