INT343628

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Context Info
Confidence 0.19
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 0.40

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (Cav2) mitochondrion organization (Cav2) intracellular (Cav2)
protein complex (Cav2) cytoplasm (Cav2) cytosol (Cav2)
Cav2 (Mus musculus)
Pain Link Frequency Relevance Heat
Calcium channel 14 100.00 Very High Very High Very High
Pyramidal cell 15 93.60 High High
Glutamate receptor 6 89.52 High High
Glutamate 7 75.00 Quite High
Action potential 22 71.76 Quite High
addiction 8 65.20 Quite High
Hippocampus 4 37.68 Quite Low
Neuronal excitability 3 25.00 Low Low
Neurotransmitter 2 25.00 Low Low
antagonist 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Targeted Disruption 7 5.00 Very Low Very Low Very Low
Disease 2 5.00 Very Low Very Low Very Low
Sprains And Strains 1 5.00 Very Low Very Low Very Low
Intellectual Impairment 1 5.00 Very Low Very Low Very Low
Neurological Disease 1 5.00 Very Low Very Low Very Low
Epilepsy 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Here we show that upregulation of Cav2.3 R-type calcium channels is responsible for a component of the ADP lasting several hundred milliseconds.
Regulation (responsible) of Positive_regulation (upregulation) of Cav2 associated with calcium channel
1) Confidence 0.19 Published 2010 Journal PLoS Biology Section Abstract Doc Link PMC2982802 Disease Relevance 0 Pain Relevance 0.40

General Comments

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