INT343712

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Context Info
Confidence 0.50
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.93
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Nos3) Golgi apparatus (Nos3) cytoplasm (Nos3)
signal transduction (Nos3) oxidoreductase activity (Nos3) nucleolus (Nos3)
Anatomy Link Frequency
vasculature 2
Nos3 (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 6 5.00 Very Low Very Low Very Low
alcohol 6 5.00 Very Low Very Low Very Low
ketamine 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
fibrosis 2 5.00 Very Low Very Low Very Low
Immobilon 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 10 93.92 High High
Pulmonary Disease 10 92.96 High High
Hypertension 54 89.52 High High
Hypoxia 30 88.44 High High
Stress 14 78.04 Quite High
Targeted Disruption 6 5.00 Very Low Very Low Very Low
INFLAMMATION 6 5.00 Very Low Very Low Very Low
Sepsis 4 5.00 Very Low Very Low Very Low
Asthma 4 5.00 Very Low Very Low Very Low
Cardiomegaly 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This increase in eNOS protein/activity levels is thought to be responsible for the protective effects on the vasculature.
Regulation (responsible) of Positive_regulation (increase) of eNOS in vasculature
1) Confidence 0.50 Published 2010 Journal PLoS ONE Section Body Doc Link PMC2982841 Disease Relevance 0.60 Pain Relevance 0
Estrogen has been shown to influence the expression and activity of endothelial nitric oxide synthase (eNOS) which is associated with increased S-nitrosothiol production.
Regulation (influence) of Positive_regulation (activity) of eNOS
2) Confidence 0.25 Published 2010 Journal PLoS ONE Section Abstract Doc Link PMC2982841 Disease Relevance 0.33 Pain Relevance 0

General Comments

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