INT343849

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Context Info
Confidence 0.04
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.32
Pain Relevance 0.21

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (FAS, TLR4) signal transduction (FAS, TLR4) plasma membrane (FAS, TLR4)
aging (FAS) intracellular (TLR4) protein complex assembly (FAS)
FAS (Homo sapiens)
TLR4 (Homo sapiens)
Pain Link Frequency Relevance Heat
antagonist 2 99.20 Very High Very High Very High
Inflammatory marker 1 82.32 Quite High
cytokine 47 68.72 Quite High
Inflammatory mediators 3 66.08 Quite High
Inflammatory response 15 59.76 Quite High
agonist 54 21.76 Low Low
Inflammation 66 5.00 Very Low Very Low Very Low
fibrosis 18 5.00 Very Low Very Low Very Low
chemokine 10 5.00 Very Low Very Low Very Low
tolerance 7 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 80 82.32 Quite High
Adhesions 7 63.60 Quite High
Injury 31 50.00 Quite Low
Stress 9 40.28 Quite Low
Shock 2 21.40 Low Low
Cirrhosis 61 5.00 Very Low Very Low Very Low
Fibrosis 24 5.00 Very Low Very Low Very Low
Apoptosis 13 5.00 Very Low Very Low Very Low
Infection 12 5.00 Very Low Very Low Very Low
Reperfusion Injury 11 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Removal of these acylated SFAs from lipid A not only results in complete loss of endotoxic activity, but also makes the deacylated lipid A act as an antagonist to native lipid A, suggesting that the FAs that are acylated in lipid A play a crucial role in ligand recognition and receptor activation for TLR4.
FAs Binding (recognition) of TLR4 associated with antagonist
1) Confidence 0.04 Published 2010 Journal Fibrogenesis Tissue Repair Section Body Doc Link PMC2984459 Disease Relevance 0.32 Pain Relevance 0.21

General Comments

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