INT34473

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Context Info
Confidence 0.03
First Reported 1987
Last Reported 1987
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.07
Pain Relevance 1.23

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transport (Slc3a1) carbohydrate metabolic process (Slc3a1) cell-cell signaling (Pomc)
cytoplasm (Pomc) extracellular space (Pomc) generation of precursor metabolites and energy (Pomc)
Pomc (Rattus norvegicus)
Slc3a1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Enkephalin 6 99.68 Very High Very High Very High
antagonist 2 97.64 Very High Very High Very High
narcan 4 97.20 Very High Very High Very High
agonist 3 96.08 Very High Very High Very High
Opioid 2 86.68 High High
Dynorphin 1 82.08 Quite High
opioid receptor 1 75.00 Quite High
Potency 1 73.04 Quite High
noradrenaline 2 62.16 Quite High
adenocard 1 60.80 Quite High
Disease Link Frequency Relevance Heat
Increased Venous Pressure Under Development 2 65.84 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Whereas the effects of beta-endorphin, DAGO and [D-Ala2,D-Leu5]enkephalin could be reduced by the mu-preferential antagonist naloxone, the effects of ethylketocyclazocine and U-50488 were not changed.
beta-endorphin Regulation (effects) of D-Ala2 associated with antagonist, narcan and enkephalin
1) Confidence 0.03 Published 1987 Journal Naunyn Schmiedebergs Arch. Pharmacol. Section Abstract Doc Link 3037389 Disease Relevance 0.07 Pain Relevance 1.23

General Comments

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