INT347807

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Context Info
Confidence 0.05
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 1.57
Pain Relevance 0.13

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Golgi apparatus (GLA, A4GALT) small molecule metabolic process (GLA) extracellular region (GLA)
transferase activity, transferring glycosyl groups (A4GALT) plasma membrane organization (A4GALT) lysosome (GLA)
Anatomy Link Frequency
endothelial cells 3
smooth muscle cells 1
GLA (Homo sapiens)
A4GALT (Homo sapiens)
Pain Link Frequency Relevance Heat
Pain 6 63.76 Quite High
Potency 4 5.00 Very Low Very Low Very Low
Calcium channel 4 5.00 Very Low Very Low Very Low
palliative 2 5.00 Very Low Very Low Very Low
carbamazepine 2 5.00 Very Low Very Low Very Low
Analgesic 2 5.00 Very Low Very Low Very Low
Neuropathic pain 2 5.00 Very Low Very Low Very Low
Pain management 2 5.00 Very Low Very Low Very Low
Gabapentin 2 5.00 Very Low Very Low Very Low
Bioavailability 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 14 94.04 High High
Lysosome Storage Disease 12 89.12 High High
Fabry Disease 56 86.80 High High
Pain 8 63.76 Quite High
Angiokeratoma 4 58.32 Quite High
Coronary Heart Disease 2 54.72 Quite High
Myocardial Infarction 2 52.64 Quite High
Stroke 6 50.40 Quite High
Gauchers Disease 4 5.00 Very Low Very Low Very Low
Pompes Disease 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
-galactosidase A (GLA), which results in the accumulation of the glycosphingolipid, globotriaosylceramide (Gb3), in different cells and organs, notably in endothelial cells and smooth muscle cells of blood vessels [1].
GLA Positive_regulation (results) of Positive_regulation (accumulation) of Gb3 in endothelial cells
1) Confidence 0.05 Published 2010 Journal Current Chemical Genomics Section Body Doc Link PMC2995157 Disease Relevance 0.78 Pain Relevance 0.06
-galactosidase A (GLA), which results in the accumulation of the glycosphingolipid, globotriaosylceramide (Gb3), in different cells and organs, notably in endothelial cells and smooth muscle cells of blood vessels [1].
GLA Positive_regulation (results) of in smooth muscle cells Positive_regulation (accumulation) of Gb3 in endothelial cells
2) Confidence 0.02 Published 2010 Journal Current Chemical Genomics Section Body Doc Link PMC2995157 Disease Relevance 0.78 Pain Relevance 0.06

General Comments

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