INT348213

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Context Info
Confidence 0.32
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.58
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

nucleoplasm (Akt2) transport (Akt2) mitochondrion (Akt2)
carbohydrate metabolic process (Akt2) cytoplasm (Akt2) cytosol (Akt2)
Anatomy Link Frequency
endothelial cells 1
Akt2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
ischemia 9 5.00 Very Low Very Low Very Low
agonist 9 5.00 Very Low Very Low Very Low
anesthesia 4 5.00 Very Low Very Low Very Low
isoflurane 2 5.00 Very Low Very Low Very Low
ketamine 1 5.00 Very Low Very Low Very Low
Potency 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Catecholamine 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diabetes Mellitus 9 83.84 Quite High
Myocardial Infarction 104 82.72 Quite High
Cardiovascular Disease 4 67.20 Quite High
Insulin Resistance 12 63.44 Quite High
Hyperglycemia 4 61.60 Quite High
Coronary Artery Disease 1 36.40 Quite Low
Body Weight 13 28.68 Quite Low
Targeted Disruption 3 12.96 Low Low
Cv Unclassified Under Development 9 5.00 Very Low Very Low Very Low
Weight Loss 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Indeed, endothelial cells had a substantial proliferative response to exenatide treatment and this response was mediated by activation of both the AKT2/eNOS and the PKA/PI3K signal transduction pathways.
Positive_regulation (activation) of AKT2 in endothelial cells
1) Confidence 0.32 Published 2010 Journal Cardiovasc Diabetol Section Body Doc Link PMC2996354 Disease Relevance 0.58 Pain Relevance 0

General Comments

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