INT349031

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Context Info
Confidence 0.36
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.23
Pain Relevance 0.09

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

aging (Epo, Akt1) nucleoplasm (Akt1) transport (Akt1)
enzyme binding (Akt1) carbohydrate metabolic process (Akt1) cytoplasm (Akt1)
Anatomy Link Frequency
endothelial cells 1
Epo (Rattus norvegicus)
Akt1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Hippocampus 3 74.12 Quite High
ischemia 10 62.40 Quite High
Neurotransmitter 1 51.56 Quite High
excitatory amino acid 1 47.60 Quite Low
bDMF 4 40.96 Quite Low
Demyelination 1 28.72 Quite Low
antagonist 1 24.72 Low Low
Central nervous system 1 21.68 Low Low
isoflurane 3 5.00 Very Low Very Low Very Low
Spinal cord 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Injury 2 100.00 Very High Very High Very High
Stress 1 64.40 Quite High
Cv Unclassified Under Development 8 62.40 Quite High
Brain Hemorrhage 2 50.00 Quite Low
Demyelinating Disease 1 28.72 Quite Low
Encephalitis 1 27.76 Quite Low
Infarction 9 5.00 Very Low Very Low Very Low
Stroke 6 5.00 Very Low Very Low Very Low
Convulsion 3 5.00 Very Low Very Low Very Low
Middle Cerebral Artery Infarction 3 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In addition, as in the case with erythroid precusor cells, EPO also seemed to be a survival factor for endothelial cells through the prevention of cell injury and DNA fragmentation by activating AKT1 and inhibiting cytochrome c release and caspase activity (Chong et al., 2002).
EPO Positive_regulation (activating) of AKT1 in endothelial cells associated with injury
1) Confidence 0.36 Published 2010 Journal Anatomy & Cell Biology Section Body Doc Link PMC2998786 Disease Relevance 0.23 Pain Relevance 0.09

General Comments

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