INT349823

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Context Info
Confidence 0.33
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.53
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

molecular_function (Atg9b) cellular_component (Atg9b) cytoplasm (Atg9b)
Anatomy Link Frequency
thoracic 1
Atg9b (Rattus norvegicus)
Pain Link Frequency Relevance Heat
neurotrophin 3 1 79.40 Quite High
Nerve growth factor 1 32.08 Quite Low
intrathecal 1 19.44 Low Low
Spinal cord 17 5.00 Very Low Very Low Very Low
Inflammation 8 5.00 Very Low Very Low Very Low
dorsal root ganglion 6 5.00 Very Low Very Low Very Low
Inflammatory response 6 5.00 Very Low Very Low Very Low
Neuropeptide 5 5.00 Very Low Very Low Very Low
Central nervous system 2 5.00 Very Low Very Low Very Low
cerebral cortex 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 24 99.60 Very High Very High Very High
Spinal Cord Injury 29 97.72 Very High Very High Very High
Death 12 46.88 Quite Low
Injury 35 5.00 Very Low Very Low Very Low
INFLAMMATION 14 5.00 Very Low Very Low Very Low
Spinal Injuries 8 5.00 Very Low Very Low Very Low
Ganglion Cysts 7 5.00 Very Low Very Low Very Low
Contusions 6 5.00 Very Low Very Low Very Low
Stress 5 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The nominal down regulation of Pycard and Casp2, lack of a significant effect on the expression of Atg9a, Atg9b, Bax, or Tp53 and lack of any TUNEL immunostaining post-SCI, strongly suggests that LDPT neurons do not undergoing apoptosis during the first month post-thoracic SCI.
Gene_expression (expression) of Atg9b in thoracic associated with spinal cord injury and apoptosis
1) Confidence 0.33 Published 2010 Journal BMC Neurosci Section Body Doc Link PMC3001741 Disease Relevance 0.53 Pain Relevance 0.04

General Comments

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