INT349858

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Context Info
Confidence 0.09
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.32
Pain Relevance 0.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Ngfr, Ntrk2) endosome (Ntrk2) cell differentiation (Ngfr)
nuclear envelope (Ngfr) aging (Ntrk2) Golgi apparatus (Ngfr)
Anatomy Link Frequency
sympathetic 1
Ngfr (Rattus norvegicus)
Ntrk2 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Nerve growth factor 1 87.52 High High
intrathecal 1 74.88 Quite High
Inflammatory response 6 51.64 Quite High
neurotrophin 3 1 33.84 Quite Low
Spinal cord 17 24.12 Low Low
Inflammation 8 5.00 Very Low Very Low Very Low
dorsal root ganglion 6 5.00 Very Low Very Low Very Low
Neuropeptide 5 5.00 Very Low Very Low Very Low
Central nervous system 2 5.00 Very Low Very Low Very Low
cerebral cortex 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Death 12 99.08 Very High Very High Very High
Apoptosis 24 97.48 Very High Very High Very High
Spinal Cord Injury 29 76.04 Quite High
INFLAMMATION 14 51.28 Quite High
Frailty 2 42.88 Quite Low
Injury 35 13.16 Low Low
Spinal Injuries 8 5.00 Very Low Very Low Very Low
Ganglion Cysts 7 5.00 Very Low Very Low Very Low
Contusions 6 5.00 Very Low Very Low Very Low
Stress 5 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
For instance, when sympathetic neurons, expressing p75 and TrkA surface receptors, were presented with the neurotrophic molecule BDNF, subsequent binding of BDNF to the p75NTR without binding to TrkB ultimately led to the death of the neurons via p75NTR induced apoptosis [76,77].
p75NTR Binding (binding) of TrkB in sympathetic associated with apoptosis and death
1) Confidence 0.09 Published 2010 Journal BMC Neurosci Section Body Doc Link PMC3001741 Disease Relevance 0.32 Pain Relevance 0.11

General Comments

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