INT350247

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Context Info
Confidence 0.36
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 2
Disease Relevance 0.99
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (Fabp4) lipid particle (Fabp4) nucleus (Fabp4)
cytoplasm (Fabp4)
Anatomy Link Frequency
fat 2
Fabp4 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
tolerance 6 26.68 Quite Low
metalloproteinase 6 5.00 Very Low Very Low Very Low
dexamethasone 2 5.00 Very Low Very Low Very Low
anesthesia 2 5.00 Very Low Very Low Very Low
alcohol 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Hypertrophy 8 96.08 Very High Very High Very High
Obesity 100 92.36 High High
Hyperlipidemia 4 64.92 Quite High
Body Weight 12 63.80 Quite High
Hypertension 4 53.76 Quite High
Cancer 6 52.40 Quite High
Diabetes Mellitus 8 38.96 Quite Low
Impaired Glucose Tolerance 6 26.68 Quite Low
Disorder Of Lipid Metabolism 12 5.00 Very Low Very Low Very Low
Hyperplasia 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
In the present study, the KCL group reduced accumulation of fat and mRNA expression of aP2 and PPAR?
Transcription (expression) of aP2 in fat
1) Confidence 0.36 Published 2010 Journal Lipids Health Dis Section Body Doc Link PMC3002919 Disease Relevance 0.47 Pain Relevance 0
Furthermore, KCL significantly inhibited pancreatic lipase activity in vitro, and also significantly inhibited fat accumulation and mRNA expression of fatty acid binding protein 2 (aP2) and peroxisome proliferator-activated ?
Transcription (expression) of aP2 in fat
2) Confidence 0.36 Published 2010 Journal Lipids Health Dis Section Abstract Doc Link PMC3002919 Disease Relevance 0.52 Pain Relevance 0

General Comments

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