INT350344

From wiki-pain
Jump to: navigation, search
Context Info
Confidence 0.10
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 2
Disease Relevance 0
Pain Relevance 0.79

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Kcnn2)
Kcnn2 (Mus musculus)
Ltp (Mus musculus)
Pain Link Frequency Relevance Heat
long-term potentiation 126 100.00 Very High Very High Very High
Calcium channel 2 91.00 High High
Hippocampus 20 83.60 Quite High
potassium channel 12 73.52 Quite High
noradrenaline 2 14.68 Low Low
agonist 44 5.00 Very Low Very Low Very Low
Pyramidal cell 30 5.00 Very Low Very Low Very Low
antagonist 14 5.00 Very Low Very Low Very Low
Action potential 10 5.00 Very Low Very Low Very Low
depression 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Cognitive Disorder 14 5.00 Very Low Very Low Very Low
Depression 6 5.00 Very Low Very Low Very Low
Disease 6 5.00 Very Low Very Low Very Low
Camurati-engelmann Disease 2 5.00 Very Low Very Low Very Low
Targeted Disruption 2 5.00 Very Low Very Low Very Low
Schizophrenia 2 5.00 Very Low Very Low Very Low
Ganglion Cysts 2 5.00 Very Low Very Low Very Low
Spontaneous Rupture 2 5.00 Very Low Very Low Very Low
Disseminated Intravascular Coagulation 2 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Studies have also shown that blocking SK channels facilitates the induction of LTP (Behnisch and Reymann, 1998; Stackman et al., 2002) and that genetic overexpression of SK2 channels inhibits LTP induction and hippocampal-dependent learning (Hammond et al., 2006).
Gene_expression (overexpression) of SK2 Negative_regulation (inhibits) of Positive_regulation (induction) of LTP associated with long-term potentiation
1) Confidence 0.10 Published 2010 Journal Neuron Section Body Doc Link PMC3003154 Disease Relevance 0 Pain Relevance 0.49
The application of apamin has previously been shown to facilitate the induction of LTP (Behnisch and Reymann, 1998; Lin et al., 2008; Ngo-Anh et al., 2005; Stackman et al., 2002) and genetic overexpression of SK2 channels inhibits the induction of LTP (Hammond et al., 2006).
Gene_expression (overexpression) of SK2 Negative_regulation (inhibits) of Positive_regulation (induction) of LTP associated with long-term potentiation
2) Confidence 0.10 Published 2010 Journal Neuron Section Body Doc Link PMC3003154 Disease Relevance 0 Pain Relevance 0.30

General Comments

This test has worked.

Personal tools
Namespaces

Variants
Actions
Navigation
Toolbox