INT350508

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Context Info
Confidence 0.54
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.50
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (ABL1) protein modification process (ABL1) mitochondrion (ABL1)
nucleolus (ABL1) nucleus (ABL1) DNA binding (ABL1)
ABL1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 3 5.00 Very Low Very Low Very Low
palliative 1 5.00 Very Low Very Low Very Low
chemokine 1 5.00 Very Low Very Low Very Low
Nicotine 1 5.00 Very Low Very Low Very Low
Inflammation 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Myeloid Leukemia 25 98.20 Very High Very High Very High
Stress 1 81.92 Quite High
Cancer 9 59.76 Quite High
Apoptosis 21 52.96 Quite High
Myeloproliferative Disorder 45 25.76 Quite Low
Retinoblastoma 1 9.28 Low Low
Myelodysplastic Syndromes 38 5.00 Very Low Very Low Very Low
Myelofibrosis 30 5.00 Very Low Very Low Very Low
Chronic Neutrophilic Leukemia 7 5.00 Very Low Very Low Very Low
Disease 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Reduction of BCR-ABL levels post-HDACi treatment, observed in most cases of HDACi treated CML cells, appears to be a consequence of disrupted Hsp90 chaperone function and proteosomal degradation of BCR-ABL [12, 65, 67, 76].
Protein_catabolism (degradation) of BCR-ABL associated with myeloid leukemia
1) Confidence 0.54 Published 2010 Journal Invest New Drugs Section Body Doc Link PMC3003795 Disease Relevance 0.50 Pain Relevance 0

General Comments

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