INT350517

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Context Info
Confidence 0.23
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.53
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (ABL1) protein modification process (ABL1) mitochondrion (ABL1)
nucleolus (ABL1) nucleus (ABL1) DNA binding (ABL1)
ABL1 (Homo sapiens)
Pain Link Frequency Relevance Heat
cytokine 3 5.00 Very Low Very Low Very Low
palliative 1 5.00 Very Low Very Low Very Low
chemokine 1 5.00 Very Low Very Low Very Low
Nicotine 1 5.00 Very Low Very Low Very Low
Inflammation 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 21 99.32 Very High Very High Very High
Cancer 9 91.60 High High
Blast Crisis 1 73.76 Quite High
Myeloid Leukemia 25 72.00 Quite High
Philadelphia Chromosome 1 56.68 Quite High
Myeloproliferative Disorder 45 49.04 Quite Low
Chronic Myeloid Leukemia 4 48.44 Quite Low
Myelodysplastic Syndromes 38 5.00 Very Low Very Low Very Low
Myelofibrosis 30 5.00 Very Low Very Low Very Low
Chronic Neutrophilic Leukemia 7 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Recent studies indicate that HDACi are capable of attenuating the expression of BCR-ABL and of inducing apoptosis in both imatinib sensitive and resistant cell lines [64–69].
Negative_regulation (attenuating) of Gene_expression (expression) of BCR-ABL associated with apoptosis
1) Confidence 0.23 Published 2010 Journal Invest New Drugs Section Body Doc Link PMC3003795 Disease Relevance 0.53 Pain Relevance 0

General Comments

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