INT350897

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Context Info
Confidence 0.18
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 1.19
Pain Relevance 0.04

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (ABL1) protein modification process (ABL1) mitochondrion (ABL1)
nucleolus (ABL1) nucleus (ABL1) DNA binding (ABL1)
ABL1 (Homo sapiens)
Pain Link Frequency Relevance Heat
rheumatoid arthritis 1 88.52 High High
cytokine 20 5.00 Very Low Very Low Very Low
addiction 1 5.00 Very Low Very Low Very Low
corticosteroid 1 5.00 Very Low Very Low Very Low
cINOD 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Disease 10 98.86 Very High Very High Very High
Myeloid Leukemia 27 95.48 Very High Very High Very High
Systemic Lupus Erythematosus 2 89.72 High High
Rheumatoid Arthritis 1 88.52 High High
Bullous Skin Disease 1 87.64 High High
Nephrotoxicity 1 86.80 High High
Thrombocytopenia 1 86.16 High High
Thyroid Disease 2 84.80 Quite High
Hemolytic Anemia 1 83.80 Quite High
Increased Venous Pressure Under Development 4 83.08 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The first of these disease-tailored products has been imatinib mesylate (IM) which blocks the ATP-binding pocket on the BCR-ABL tyrosine-kinase and thus prevents the activation of this enzyme which plays the key role in the pathogenesis of CML [13].
Negative_regulation (blocks) of BCR-ABL Binding (binding) of associated with myeloid leukemia and disease
1) Confidence 0.18 Published 2010 Journal Clinical and Developmental Immunology Section Body Doc Link PMC3004381 Disease Relevance 1.19 Pain Relevance 0.04

General Comments

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