INT351002

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Context Info
Confidence 0.00
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 5
Disease Relevance 3.65
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

intracellular (Adh7) signal transducer activity (Mrgpre) cytoplasm (Adh7)
signal transduction (Mrgpre) oxidoreductase activity (Adh7) nucleolus (Adh7)
Anatomy Link Frequency
blood 1
Adh7 (Mus musculus)
Mrgpre (Mus musculus)
Pain Link Frequency Relevance Heat
agonist 25 5.00 Very Low Very Low Very Low
cva 10 5.00 Very Low Very Low Very Low
Pain 5 5.00 Very Low Very Low Very Low
alcohol 5 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Apoptosis 15 98.98 Very High Very High Very High
Metastasis 25 98.12 Very High Very High Very High
Cancer 130 97.66 Very High Very High Very High
Prostate Cancer 280 97.32 Very High Very High Very High
Hypoxia 5 93.34 High High
Recurrence 25 71.84 Quite High
Disease 95 5.00 Very Low Very Low Very Low
Osteoporosis 65 5.00 Very Low Very Low Very Low
Hot Flashes 40 5.00 Very Low Very Low Very Low
Metabolic Syndrome 35 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
First, the field dimensions used in RT can be smaller, thereby allowing administration of a higher total dose without increased side effects to healthy tissue.19 Data from numerous studies indicate that neoadjuvant HT results in substantial tumor volume reduction, ranging from 30% to 40%.20 Another postulated mechanism refers to possible enhanced oxygenation (and related increased loco-regional RT effectiveness) of the hypoxic PC cells by improving blood flow with decreasing interstitial pressure when the total amount of cancer cells in the tumor is diminished by ADT.21 It is also plausible that apoptosis induced by HT could affect cancer cells in which apoptosis was not activated by RT.22 The apoptotic mechanism could also be relevant for the systemic interaction of ADT–EBRT by prevention of the subsequent distant micro metastases.
ADT Binding (interaction) of EBRT in blood associated with hypoxia, cancer, prostate cancer, apoptosis and metastasis
1) Confidence 0.00 Published 2010 Journal Cancer management and research Section Body Doc Link PMC3004567 Disease Relevance 1.24 Pain Relevance 0
The possible mechanism of ADT–EBRT interaction in that study was the diminished growth velocity of the surviving PC cells after neoadjuvant ADT.
ADT Binding (interaction) of EBRT associated with prostate cancer
2) Confidence 0.00 Published 2010 Journal Cancer management and research Section Body Doc Link PMC3004567 Disease Relevance 0.63 Pain Relevance 0
Other more clinical observations shed some light on ADT–EBRT interactions.
ADT Binding (interactions) of EBRT
3) Confidence 0.00 Published 2010 Journal Cancer management and research Section Body Doc Link PMC3004567 Disease Relevance 0.58 Pain Relevance 0
The possible mechanism of ADT–EBRT interaction in that study was the diminished growth velocity of the surviving PC cells after neoadjuvant ADT.
ADT Binding (interaction) of EBRT associated with prostate cancer
4) Confidence 0.00 Published 2010 Journal Cancer management and research Section Body Doc Link PMC3004567 Disease Relevance 0.63 Pain Relevance 0
Other more clinical observations shed some light on ADT–EBRT interactions.
ADT Binding (interactions) of EBRT
5) Confidence 0.00 Published 2010 Journal Cancer management and research Section Body Doc Link PMC3004567 Disease Relevance 0.58 Pain Relevance 0

General Comments

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