INT351013

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.76
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell proliferation (ISG20) nucleoplasm (ISG20) extracellular region (PROK1)
RNA binding (ISG20) nucleus (ISG20) intracellular (ISG20)
Anatomy Link Frequency
T-cells 1
PROK1 (Homo sapiens)
ISG20 (Homo sapiens)
Pain Link Frequency Relevance Heat
pruritus 4 5.00 Very Low Very Low Very Low
cytokine 3 5.00 Very Low Very Low Very Low
Paracetamol 3 5.00 Very Low Very Low Very Low
Pain 2 5.00 Very Low Very Low Very Low
Antihistamine 2 5.00 Very Low Very Low Very Low
dexamethasone 1 5.00 Very Low Very Low Very Low
depression 1 5.00 Very Low Very Low Very Low
vincristine 1 5.00 Very Low Very Low Very Low
tolerance 1 5.00 Very Low Very Low Very Low
rheumatoid arthritis 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Diphtheria 7 100.00 Very High Very High Very High
Disease 20 99.44 Very High Very High Very High
Lymphoproliferative Disease 1 94.88 High High
Lymphatic System Cancer 39 93.36 High High
Malignant Neoplastic Disease 5 88.16 High High
Mycosis Fungoides 12 5.00 Very Low Very Low Very Low
Syndrome 12 5.00 Very Low Very Low Very Low
Vomiting 6 5.00 Very Low Very Low Very Low
Cancer 6 5.00 Very Low Very Low Very Low
Leukemia 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
This fusion protein toxin combines the IL2 protein with diphtheria toxin, and targets the CD25 subunit of the IL2 receptor, resulting in the unique delivery of a cytocidal agent to CD-25 bearing T-cells.
protein toxin Regulation (targets) of CD25 in T-cells associated with diphtheria
1) Confidence 0.01 Published 2010 Journal Cancer management and research Section Abstract Doc Link PMC3004568 Disease Relevance 0.76 Pain Relevance 0

General Comments

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