INT353125

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Context Info
Confidence 0.04
First Reported 2007
Last Reported 2007
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 6
Disease Relevance 5.92
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (VEGFA, Kdr) extracellular space (VEGFA) growth (VEGFA)
extracellular region (VEGFA) proteinaceous extracellular matrix (VEGFA) plasma membrane (Kdr)
VEGFA (Homo sapiens)
Kdr (Mus musculus)
Pain Link Frequency Relevance Heat
cva 48 5.00 Very Low Very Low Very Low
palliative 18 5.00 Very Low Very Low Very Low
headache 12 5.00 Very Low Very Low Very Low
cytokine 6 5.00 Very Low Very Low Very Low
Pain 6 5.00 Very Low Very Low Very Low
Calcium channel 6 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Non-small-cell Lung Cancer 768 96.72 Very High Very High Very High
Cancer 462 93.60 High High
Metastasis 60 87.88 High High
Lung Cancer 138 67.40 Quite High
Hypoxia 30 54.16 Quite High
Edema 6 26.36 Quite Low
Disease 198 5.00 Very Low Very Low Very Low
Hemorrhage 174 5.00 Very Low Very Low Very Low
Pressure And Volume Under Development 78 5.00 Very Low Very Low Very Low
Disease Progression 66 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
inhibiting VEGF itself using neutralizing anti-VEGF monoclonal antibodies or soluble receptorsVEGF receptor (VEGFR) blockade using anti-VEGFR monoclonal antibodiesinhibiting VEGFR signalling with small molecule VEGFR tyrosine kinase inhibitors.


Negative_regulation (inhibitors) of VEGF Binding (signalling) of VEGFR
1) Confidence 0.04 Published 2007 Journal Core Evidence Section Body Doc Link PMC3012552 Disease Relevance 1.01 Pain Relevance 0
inhibiting VEGF itself using neutralizing anti-VEGF monoclonal antibodies or soluble receptorsVEGF receptor (VEGFR) blockade using anti-VEGFR monoclonal antibodiesinhibiting VEGFR signalling with small molecule VEGFR tyrosine kinase inhibitors.


Negative_regulation (inhibitors) of VEGF Binding (signalling) of VEGFR
2) Confidence 0.04 Published 2007 Journal Core Evidence Section Body Doc Link PMC3012552 Disease Relevance 1.01 Pain Relevance 0
inhibiting VEGF itself using neutralizing anti-VEGF monoclonal antibodies or soluble receptorsVEGF receptor (VEGFR) blockade using anti-VEGFR monoclonal antibodiesinhibiting VEGFR signalling with small molecule VEGFR tyrosine kinase inhibitors.


Negative_regulation (antibodiesinhibiting) of VEGF Binding (signalling) of VEGFR
3) Confidence 0.04 Published 2007 Journal Core Evidence Section Body Doc Link PMC3012552 Disease Relevance 0.94 Pain Relevance 0
inhibiting VEGF itself using neutralizing anti-VEGF monoclonal antibodies or soluble receptorsVEGF receptor (VEGFR) blockade using anti-VEGFR monoclonal antibodiesinhibiting VEGFR signalling with small molecule VEGFR tyrosine kinase inhibitors.


Negative_regulation (antibodiesinhibiting) of VEGF Binding (signalling) of VEGFR
4) Confidence 0.04 Published 2007 Journal Core Evidence Section Body Doc Link PMC3012552 Disease Relevance 1.00 Pain Relevance 0
inhibiting VEGF itself using neutralizing anti-VEGF monoclonal antibodies or soluble receptorsVEGF receptor (VEGFR) blockade using anti-VEGFR monoclonal antibodiesinhibiting VEGFR signalling with small molecule VEGFR tyrosine kinase inhibitors.


Negative_regulation (inhibitors) of VEGF Binding (signalling) of anti-VEGFR
5) Confidence 0.03 Published 2007 Journal Core Evidence Section Body Doc Link PMC3012552 Disease Relevance 1.01 Pain Relevance 0
inhibiting VEGF itself using neutralizing anti-VEGF monoclonal antibodies or soluble receptorsVEGF receptor (VEGFR) blockade using anti-VEGFR monoclonal antibodiesinhibiting VEGFR signalling with small molecule VEGFR tyrosine kinase inhibitors.


Negative_regulation (antibodiesinhibiting) of VEGF Binding (signalling) of anti-VEGFR
6) Confidence 0.03 Published 2007 Journal Core Evidence Section Body Doc Link PMC3012552 Disease Relevance 0.94 Pain Relevance 0

General Comments

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