INT353331

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Context Info
Confidence 0.08
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.78
Pain Relevance 0.44

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

peptidase activity (Elane) plasma membrane (F2r) molecular_function (Elane)
cellular_component (Elane) signal transducer activity (F2r) biological_process (Elane)
Anatomy Link Frequency
epithelial cells 1
Elane (Rattus norvegicus)
F2r (Rattus norvegicus)
Pain Link Frequency Relevance Heat
interstitial cystitis 3 99.92 Very High Very High Very High
Inflammation 22 76.96 Quite High
agonist 5 69.12 Quite High
Inflammatory stimuli 5 58.24 Quite High
lidocaine 9 5.00 Very Low Very Low Very Low
cytokine 4 5.00 Very Low Very Low Very Low
halothane 3 5.00 Very Low Very Low Very Low
substance P 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Thoracotomy 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Interstitial Cystitis 3 99.92 Very High Very High Very High
Apoptosis 1 99.04 Very High Very High Very High
Cystitis 4 90.04 High High
INFLAMMATION 26 76.96 Quite High
Cancer 4 5.00 Very Low Very Low Very Low
Pain 1 5.00 Very Low Very Low Very Low
Overdose 1 5.00 Very Low Very Low Very Low
Neurogenic Inflammation 1 5.00 Very Low Very Low Very Low
Peripheral Arterial Disease 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Activation of PAR1 receptors by neutrophil elastase was reported to induce apoptosis in lung epithelial cells [36] while activation of PAR1 and PAR2 receptors were shown to increase epithelial permeability in intestinal epithelia [37].
neutrophil elastase Positive_regulation (Activation) of PAR1 in epithelial cells associated with apoptosis
1) Confidence 0.08 Published 2010 Journal PLoS ONE Section Body Doc Link PMC3013117 Disease Relevance 0.78 Pain Relevance 0.44

General Comments

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