INT35360

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Context Info
Confidence 0.22
First Reported 1988
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 3
Total Number 7
Disease Relevance 3.55
Pain Relevance 0.81

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

Anatomy Link Frequency
lung 1
Ve (Mus musculus)
Pain Link Frequency Relevance Heat
narcan 6 99.46 Very High Very High Very High
antagonist 2 98.98 Very High Very High Very High
Morphine 4 93.28 High High
depression 4 92.52 High High
Opioid 3 70.12 Quite High
rapifen 2 50.00 Quite Low
Pain 5 42.72 Quite Low
fibrosis 5 40.40 Quite Low
Antiemetics 1 10.00 Low Low
cva 10 6.80 Low Low
Disease Link Frequency Relevance Heat
Increased Venous Pressure Under Development 25 99.84 Very High Very High Very High
Systemic Sclerosis 155 99.20 Very High Very High Very High
Cv Unclassified Under Development 105 98.48 Very High Very High Very High
Disease 70 97.20 Very High Very High Very High
Depression 1 92.52 High High
Heart Rate Under Development 5 92.20 High High
Pulmonary Hypertension 80 92.08 High High
Congenital Anomalies 35 86.60 High High
Left Ventricular Dysfunction 10 84.92 Quite High
Pulmonary Disease 40 80.16 Quite High

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
However, for the 1.5-6 h after antagonist, VE8 following naloxone became depressed, whereas VE8 after nalmefene remained significantly increased compared with other treatments.
Positive_regulation (increased) of VE8 associated with antagonist and narcan
1) Confidence 0.22 Published 1988 Journal Br J Anaesth Section Abstract Doc Link 3140862 Disease Relevance 0.23 Pain Relevance 0.81
Figure 5 relates the degree of lung restriction (reduced FVC) to the elevation of VE/VCO2 (Fig. 5a) and dilution of PCO2 (Fig. 5b) at the AT or VCP.
Positive_regulation (elevation) of VE in lung
2) Confidence 0.02 Published 2010 Journal PLoS ONE Section Body Doc Link PMC3001444 Disease Relevance 0.80 Pain Relevance 0
WR, and ventilation-perfusion mismatch (elevated VE/VCO2 at the AT or at the ventilatory compensation point (VCP) following AT).
Positive_regulation (elevated) of VE
3) Confidence 0.02 Published 2010 Journal PLoS ONE Section Body Doc Link PMC3001444 Disease Relevance 0.48 Pain Relevance 0
However, increased VE/VCO2 beyond that found in normal subjects [26], and simultaneously decreased PETCO2 at the AT, as well as specific changes in the patterns of these two variables as work rate is increased, strongly suggest that dead space ventilation is increased.
Positive_regulation (increased) of VE
4) Confidence 0.02 Published 2010 Journal PLoS ONE Section Body Doc Link PMC3001444 Disease Relevance 0.72 Pain Relevance 0
In less severe stages of pulmonary vascular disease, small increases in VE/VCO2 are accompanied by large decreases in PETCO2 [18] (Figure 4).
Positive_regulation (increases) of VE associated with increased venous pressure under development
5) Confidence 0.02 Published 2010 Journal PLoS ONE Section Body Doc Link PMC3001444 Disease Relevance 0.77 Pain Relevance 0
Due to its pathogenesis, VE/VCO2 should invariably be increased in patients with PV [18], [24], [25].
Positive_regulation (increased) of VE
6) Confidence 0.02 Published 2010 Journal PLoS ONE Section Body Doc Link PMC3001444 Disease Relevance 0.55 Pain Relevance 0
After diphenhydramine, the increase in VE90 to 27.2+/-9.2 l/min was not significant (P = 0.06).
Positive_regulation (increase) of VE90
7) Confidence 0.00 Published 1998 Journal Anesthesiology Section Body Doc Link 9743400 Disease Relevance 0 Pain Relevance 0

General Comments

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