INT353697

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.42
Pain Relevance 0.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Slc8a1) plasma membrane (Slc8a1) transmembrane transport (Slc8a1)
response to stress (Slc8a1)
Anatomy Link Frequency
plasma 1
NA (Homo sapiens)
Slc8a1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
nMDA receptor 33 91.52 High High
ischemia 55 74.24 Quite High
Glutamate 18 61.56 Quite High
bDMF 3 5.00 Very Low Very Low Very Low
Glutamate receptor 3 5.00 Very Low Very Low Very Low
isoflurane 3 5.00 Very Low Very Low Very Low
adenocard 2 5.00 Very Low Very Low Very Low
tetrodotoxin 1 5.00 Very Low Very Low Very Low
Central nervous system 1 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Death 5 97.62 Very High Very High Very High
Injury 9 78.44 Quite High
Cv General 4 Under Development 2 74.24 Quite High
Brain Hemorrhage 38 55.04 Quite High
Cv Unclassified Under Development 15 53.48 Quite High
Infarction 3 5.00 Very Low Very Low Very Low
Hypoxia 3 5.00 Very Low Very Low Very Low
Middle Cerebral Artery Infarction 2 5.00 Very Low Very Low Very Low
Parkinson's Disease 1 5.00 Very Low Very Low Very Low
Apoptosis 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The neurotoxic mechanism that leads to penumbra cell death in response to elevated intracellular Na+ in the current study represent the reverse operation of the plasma membrane NCX1, engaged by plasma membrane depolarization and intracellular Na+ overload through a decreased Na+/K+-ATPase.
Na Binding (engaged) of NCX1 in plasma associated with death
1) Confidence 0.01 Published 2010 Journal Anatomy & Cell Biology Section Body Doc Link PMC3015038 Disease Relevance 0.42 Pain Relevance 0.19

General Comments

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