INT353937

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Context Info
Confidence 0.52
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 4
Disease Relevance 2.44
Pain Relevance 1.01

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

extracellular space (Il21) extracellular region (Il21)
Anatomy Link Frequency
macrophages 1
endothelial cells 1
fibroblasts 1
epithelial cells 1
Il21 (Mus musculus)
Pain Link Frequency Relevance Heat
cytokine 124 91.72 High High
Crohn's disease 192 87.72 High High
Inflammation 84 81.04 Quite High
chemokine 8 37.44 Quite Low
Inflammatory mediators 8 5.00 Very Low Very Low Very Low
cINOD 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Inflammatory Bowel Disease 216 94.76 High High
Disease 212 87.72 High High
INFLAMMATION 80 81.04 Quite High
Sprains And Strains 4 68.52 Quite High
Ileal Diseases 20 43.44 Quite Low
Ileitis 4 5.00 Very Low Very Low Very Low
Targeted Disruption 4 5.00 Very Low Very Low Very Low
Colon Disease 4 5.00 Very Low Very Low Very Low
Cancer 4 5.00 Very Low Very Low Very Low
Polyps 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The invasion of extracellular bacteria into the intestinal mucosa triggers the expression of IL-23A, driving Th17 cells to release IL-17A, IL-17F, IL-21, IL-22 and IL-26, which in turn exert a number of proinflammatory effects on intestinal epithelial cells, endothelial cells, macrophages and fibroblasts [10].
Localization (release) of IL-21 in fibroblasts
1) Confidence 0.52 Published 2010 Journal BMC Immunol Section Body Doc Link PMC3016394 Disease Relevance 0.61 Pain Relevance 0.25
The invasion of extracellular bacteria into the intestinal mucosa triggers the expression of IL-23A, driving Th17 cells to release IL-17A, IL-17F, IL-21, IL-22 and IL-26, which in turn exert a number of proinflammatory effects on intestinal epithelial cells, endothelial cells, macrophages and fibroblasts [10].
Localization (release) of IL-21 in epithelial cells
2) Confidence 0.18 Published 2010 Journal BMC Immunol Section Body Doc Link PMC3016394 Disease Relevance 0.61 Pain Relevance 0.25
The invasion of extracellular bacteria into the intestinal mucosa triggers the expression of IL-23A, driving Th17 cells to release IL-17A, IL-17F, IL-21, IL-22 and IL-26, which in turn exert a number of proinflammatory effects on intestinal epithelial cells, endothelial cells, macrophages and fibroblasts [10].
Localization (release) of IL-21 in endothelial cells
3) Confidence 0.18 Published 2010 Journal BMC Immunol Section Body Doc Link PMC3016394 Disease Relevance 0.61 Pain Relevance 0.25
The invasion of extracellular bacteria into the intestinal mucosa triggers the expression of IL-23A, driving Th17 cells to release IL-17A, IL-17F, IL-21, IL-22 and IL-26, which in turn exert a number of proinflammatory effects on intestinal epithelial cells, endothelial cells, macrophages and fibroblasts [10].
Localization (release) of IL-21 in macrophages
4) Confidence 0.18 Published 2010 Journal BMC Immunol Section Body Doc Link PMC3016394 Disease Relevance 0.61 Pain Relevance 0.25

General Comments

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