INT354984

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Context Info
Confidence 0.01
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.83
Pain Relevance 0.40

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

plasma membrane (Cacna1a, Tnf) transport (Cacna1a) intracellular (Tnf)
extracellular matrix organization (Tnf) DNA binding (Cacna1a) transmembrane transport (Cacna1a)
Cacna1a (Mus musculus)
Tnf (Mus musculus)
Pain Link Frequency Relevance Heat
Inflammation 51 100.00 Very High Very High Very High
Morphine 7 100.00 Very High Very High Very High
agonist 1 100.00 Very High Very High Very High
narcan 10 5.00 Very Low Very Low Very Low
Opioid 9 5.00 Very Low Very Low Very Low
cytokine 7 5.00 Very Low Very Low Very Low
dexamethasone 4 5.00 Very Low Very Low Very Low
Antinociceptive 3 5.00 Very Low Very Low Very Low
Serotonin 2 5.00 Very Low Very Low Very Low
antagonist 2 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
INFLAMMATION 53 100.00 Very High Very High Very High
Urological Neuroanatomy 12 92.52 High High
Pressure And Volume Under Development 25 85.44 High High
Fungal Infection 1 71.36 Quite High
Ulcers 1 29.60 Quite Low
Cancer 5 28.20 Quite Low
Peritonitis 2 5.00 Very Low Very Low Very Low
Neuropathic Pain 2 5.00 Very Low Very Low Very Low
Lung Injury 2 5.00 Very Low Very Low Very Low
Hyperalgesia 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Furthermore, to clarify the LA mechanism of action, we studied the participation of pentoxifylline, a PDE5 and TNF-alpha inhibitor, and morphine, a mu and kappa agonist, on inflammatory processes, and their possible interaction with LA.
LA Binding (interaction) of TNF-alpha associated with inflammation, agonist and morphine
1) Confidence 0.01 Published 2010 Journal J Inflamm (Lond) Section Body Doc Link PMC3019217 Disease Relevance 0.83 Pain Relevance 0.40

General Comments

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