INT355245

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Context Info
Confidence 0.04
First Reported 2011
Last Reported 2011
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0
Pain Relevance 1.43

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

transport (Cacna1a) DNA binding (Cacna1a) transmembrane transport (Cacna1a)
cytoplasm (Cacna1a) cell death (Cacna1a) nucleus (Cacna1a)
Cacna1a (Mus musculus)
Pain Link Frequency Relevance Heat
amygdala 126 100.00 Very High Very High Very High
qutenza 79 100.00 Very High Very High Very High
long-term potentiation 74 99.96 Very High Very High Very High
gABA 5 95.48 Very High Very High Very High
Glutamate 5 95.16 Very High Very High Very High
Glutamate receptor 1 74.44 Quite High
antagonist 18 69.92 Quite High
GABA receptor 4 69.48 Quite High
tetrodotoxin 1 63.68 Quite High
sodium channel 1 62.84 Quite High
Disease Link Frequency Relevance Heat
Anxiety Disorder 7 5.00 Very Low Very Low Very Low
Pain 5 5.00 Very Low Very Low Very Low
Depression 3 5.00 Very Low Very Low Very Low
Targeted Disruption 2 5.00 Very Low Very Low Very Low
Nociception 2 5.00 Very Low Very Low Very Low
Hyperalgesia 1 5.00 Very Low Very Low Very Low
Epilepsy 1 5.00 Very Low Very Low Very Low
Drug Induced Neurotoxicity 1 5.00 Very Low Very Low Very Low
Cognitive Disorder 1 5.00 Very Low Very Low Very Low
Decapitation 1 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
NO and CB1 receptors are involved in mediating capsaicin-induced reduction of LA-LTP
Regulation (involved) of Localization (reduction) of LA associated with qutenza, long-term potentiation and amygdala
1) Confidence 0.04 Published 2011 Journal PLoS ONE Section Body Doc Link PMC3020947 Disease Relevance 0 Pain Relevance 1.43

General Comments

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