INT356122

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Context Info
Confidence 0.09
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 1
Disease Relevance 0.69
Pain Relevance 0.19

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

mitochondrion (Bcl2l1) intracellular (Bcl2l1) response to stress (Bcl2l1)
cytoplasm (Bcl2l1) cytosol (Bcl2l1) cell proliferation (Bcl2l1)
Anatomy Link Frequency
brain 2
Bcl2l1 (Rattus norvegicus)
Pain Link Frequency Relevance Heat
Inflammation 19 97.52 Very High Very High Very High
cytokine 14 57.24 Quite High
ischemia 14 45.16 Quite Low
Central nervous system 5 5.00 Very Low Very Low Very Low
Inflammatory response 3 5.00 Very Low Very Low Very Low
Spinal cord 2 5.00 Very Low Very Low Very Low
tolerance 2 5.00 Very Low Very Low Very Low
isoflurane 2 5.00 Very Low Very Low Very Low
anesthesia 1 5.00 Very Low Very Low Very Low
Inflammatory mediators 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Death 7 99.92 Very High Very High Very High
Apoptosis 9 99.70 Very High Very High Very High
INFLAMMATION 23 97.52 Very High Very High Very High
Injury 9 92.16 High High
Stroke 35 75.00 Quite High
Cv Unclassified Under Development 10 45.16 Quite Low
Middle Cerebral Artery Infarction 1 25.00 Low Low
Urological Neuroanatomy 6 5.00 Very Low Very Low Very Low
Reperfusion Injury 5 5.00 Very Low Very Low Very Low
Adhesions 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Such treatment also minimizes apoptotic cell death by influencing STAT-3 phosphorylation and Bcl-xL expression in the brain.
Regulation (influencing) of Phosphorylation (phosphorylation) of Bcl-xL in brain associated with apoptosis and death
1) Confidence 0.09 Published 2010 Journal Journal of Neuroinflammation Section Abstract Doc Link PMC3022819 Disease Relevance 0.69 Pain Relevance 0.19

General Comments

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