INT356313

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Context Info
Confidence 0.02
First Reported 2011
Last Reported 2011
Negated 0
Speculated 0
Reported most in Abstract
Documents 1
Total Number 4
Disease Relevance 0
Pain Relevance 2.11

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cell differentiation (Helt) extracellular space (Crh) extracellular region (Crh)
nucleus (Helt) DNA binding (Helt) response to stress (Crh)
Anatomy Link Frequency
neurons 8
Crh (Mus musculus)
Helt (Mus musculus)
Pain Link Frequency Relevance Heat
amygdala 204 100.00 Very High Very High Very High
GABAergic 64 100.00 Very High Very High Very High
gABA 96 98.48 Very High Very High Very High
withdrawal 8 98.20 Very High Very High Very High
addiction 32 96.08 Very High Very High Very High
alcohol 44 5.00 Very Low Very Low Very Low
Neurotransmitter 16 5.00 Very Low Very Low Very Low
antagonist 8 5.00 Very Low Very Low Very Low
opiate 4 5.00 Very Low Very Low Very Low
Kinase C 4 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Alcohol Addiction 32 5.00 Very Low Very Low Very Low
Anxiety Disorder 20 5.00 Very Low Very Low Very Low
Stress 8 5.00 Very Low Very Low Very Low
Fetal Death 4 5.00 Very Low Very Low Very Low
Depression 4 5.00 Very Low Very Low Very Low
Recurrence 4 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
Previously, we found that both ethanol and corticotropin releasing factor (CRF) increase GABAergic transmission in mouse and rat CeA neurons, in part by enhancing the release of GABA via activation of presynaptic CRF1 receptors.
corticotropin releasing factor Positive_regulation (increase) of Localization (transmission) of GABAergic in neurons associated with gaba, gabaergic and amygdala
1) Confidence 0.02 Published 2011 Journal Frontiers in Neuroscience Section Abstract Doc Link PMC3024005 Disease Relevance 0 Pain Relevance 0.53
Previously, we found that both ethanol and corticotropin releasing factor (CRF) increase GABAergic transmission in mouse and rat CeA neurons, in part by enhancing the release of GABA via activation of presynaptic CRF1 receptors.
corticotropin releasing factor Positive_regulation (increase) of Localization (transmission) of neurons in neurons associated with gaba, gabaergic and amygdala
2) Confidence 0.02 Published 2011 Journal Frontiers in Neuroscience Section Abstract Doc Link PMC3024005 Disease Relevance 0 Pain Relevance 0.53
Previously, we found that both ethanol and corticotropin releasing factor (CRF) increase GABAergic transmission in mouse and rat CeA neurons, in part by enhancing the release of GABA via activation of presynaptic CRF1 receptors.
CRF Positive_regulation (increase) of Localization (transmission) of GABAergic in neurons associated with gaba, gabaergic and amygdala
3) Confidence 0.02 Published 2011 Journal Frontiers in Neuroscience Section Abstract Doc Link PMC3024005 Disease Relevance 0 Pain Relevance 0.52
Previously, we found that both ethanol and corticotropin releasing factor (CRF) increase GABAergic transmission in mouse and rat CeA neurons, in part by enhancing the release of GABA via activation of presynaptic CRF1 receptors.
CRF Positive_regulation (increase) of Localization (transmission) of neurons in neurons associated with gaba, gabaergic and amygdala
4) Confidence 0.02 Published 2011 Journal Frontiers in Neuroscience Section Abstract Doc Link PMC3024005 Disease Relevance 0 Pain Relevance 0.53

General Comments

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