INT356377

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Context Info
Confidence 0.03
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.51
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytoplasm (Dmd, Mib2) endosome (Mib2) ligase activity (Mib2)
signal transducer activity (Mib2) signal transduction (Mib2) nucleus (Dmd)
Dmd (Mus musculus)
Mib2 (Mus musculus)
Pain Link Frequency Relevance Heat
fibrosis 36 5.00 Very Low Very Low Very Low
imagery 6 5.00 Very Low Very Low Very Low
Inflammation 4 5.00 Very Low Very Low Very Low
Calcium channel 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
tolerance 1 5.00 Very Low Very Low Very Low
Angina 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Muscular Dystrophy 140 99.84 Very High Very High Very High
Cardiomyopathy 63 95.08 Very High Very High Very High
Coronary Heart Disease 4 77.32 Quite High
Fibrosis 43 5.00 Very Low Very Low Very Low
Duchenne Muscular Dystrophy 20 5.00 Very Low Very Low Very Low
Dilated Cardiomyopathy 18 5.00 Very Low Very Low Very Low
Death 11 5.00 Very Low Very Low Very Low
Stress 8 5.00 Very Low Very Low Very Low
Congenital Anomalies 6 5.00 Very Low Very Low Very Low
Myocardial Infarction 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The other major treatment option is to employ molecules that allow read-through of the stop codon in the dystrophin gene that causes the loss of the dystrophin protein in DMD.
dystrophin gene Positive_regulation (causes) of Negative_regulation (loss) of dystrophin protein associated with muscular dystrophy
1) Confidence 0.03 Published 2010 Journal The Open Cardiovascular Medicine Journal Section Body Doc Link PMC3024556 Disease Relevance 0.51 Pain Relevance 0

General Comments

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