INT356378

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Context Info
Confidence 0.96
First Reported 2010
Last Reported 2010
Negated 0
Speculated 0
Reported most in Body
Documents 1
Total Number 1
Disease Relevance 0.31
Pain Relevance 0

This is a graph with borders and nodes. Maybe there is an Imagemap used so the nodes may be linking to some Pages.

cytosol (TNNI1)
Anatomy Link Frequency
cardiomyocyte 1
TNNI1 (Homo sapiens)
Pain Link Frequency Relevance Heat
fibrosis 36 15.60 Low Low
imagery 6 5.00 Very Low Very Low Very Low
Inflammation 4 5.00 Very Low Very Low Very Low
Calcium channel 2 5.00 Very Low Very Low Very Low
cytokine 1 5.00 Very Low Very Low Very Low
tolerance 1 5.00 Very Low Very Low Very Low
Angina 1 5.00 Very Low Very Low Very Low
antagonist 1 5.00 Very Low Very Low Very Low
Disease Link Frequency Relevance Heat
Stress 8 75.40 Quite High
Death 11 69.92 Quite High
Dilated Cardiomyopathy 18 58.28 Quite High
Myocardial Infarction 6 52.60 Quite High
Muscular Dystrophy 140 50.92 Quite High
Fibrosis 43 49.28 Quite Low
Duchenne Muscular Dystrophy 20 36.08 Quite Low
Muscle Weakness 2 8.24 Low Low
Cardiomyopathy 63 5.00 Very Low Very Low Very Low
Congenital Anomalies 6 5.00 Very Low Very Low Very Low

Sentences Mentioned In

Key: Protein Mutation Event Anatomy Negation Speculation Pain term Disease term
The increase of the intracellular concentration of calcium in the cardiomyocyte leads to the activation of calcium-activated proteases such as calcium-induced calpains, a family of proteases, which will degrade troponin I and thus compromise the contractions of the cardiomyocyte [13-15].
Protein_catabolism (degrade) of troponin I in cardiomyocyte
1) Confidence 0.96 Published 2010 Journal The Open Cardiovascular Medicine Journal Section Body Doc Link PMC3024556 Disease Relevance 0.31 Pain Relevance 0

General Comments

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